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Chronic Inflammatory Demyelinating Polyneuropathy Figure 1: Treatment Algorithm for Chronic Inflammatory Demyelinating Polyradiculoneuropathy


Mild to moderate impairment or disability and


no contraindications for corticosteroids treatment and


no pure motor form of CIDP


Moderate to severe impairment or disability or


contraindication for corticosteroid treatment or


pure motor form of CIDP Very mild impairment or disability


One course of IVIg 2g/kg over 2 or 5 days


Insufficient improvement or deterioration


Repeat one course of IVIg 2g/kg over 2 or 5 days


Deterioration


Consider to repeat one course of IVIg Contraindication for corticosteroids?


No


Start dexamethasone 40mg for 4 days, repeated every month for six months


Alternative: prednisone 60mg/day during 6 weeks and taper dose over 32 weeks (dexamethasone acts faster and seems to have fewer long-term side effects)


Consider to add an immunosuppressive drug, see text Yes


Wait and see Monitor without treatment


Sufficient improvement, i.e. recovery to full or nearly full ability


Wait and see Monitor without treatment


No deterioration Remission


Repeat IVIg 1g/kg once every 3 weeks. Find lowest possible dose with longest possible intervals to maintain patient’s functional status, see text


Consider to add an immunosuppressive drug, see text


Patients who fail to respond to the initial treatment or have serious side effects should be switched to IVIg or corticosteroids (dexamethasone or prednisone). Plasma exchange can be useful in patients who do not respond to corticosteroids and IVIg. Consider to add an immunosuppressive drug, see text. Autologous stem cell transplantation can be tried in therapy resistant patients


CIDP = chronic inflammatory demyelinating polyradiculoneuropathy; IVIg = intravenous immunoglobulin. Tacrolimus


Tacrolimus is an immunosuppressant that inhibits both T-lymphocyte signal transduction and interleukin (IL)-2 transcription. One case report notes that tacrolimus had a positive effect in a patient with CIDP.100 Nephrotoxicity is the most important, although reversible, side effect. Many case reports and cases series have reported tacrolimus-related polyneuropathies often resembling CIDP, however.101–104


Interferon Beta


Three case reports showed improvement after treatment with different brands of interferon beta.105–107


of 24 CIDP patients, nine patients improved significantly;108,109


In two series, including a total however,


a randomised controlled trial including 10 treatment refractory CIDP patients did not show a significant effect on predetermined impairment and disability scales. Recently, these results were confirmed in a RCT with intramuscular interferon beta-1a, which, when added to IVIg, had no effect in CIDP.110


The most commonly seen adverse events are flu-like symptoms, mild leucocytopenia and alteration of liver function. Two case reports of patients with multiple sclerosis developing CIDP after interferon beta-1b treatment have been published.111,112


These adverse events, 48


together with its doubtful efficacy and high costs, must lead to the conclusion that there is no place for interferon beta treatment in CIDP.


Interferon Alpha


Nine of 14 patients with CIDP treated with subcutaneous interferon alpha-2a three times a week for six weeks had a favourable response, although three patients later had a relapse.113 reports confirming these beneficial results.114–116


There are a few case Adverse events are


similar to those seen with interferon beta, but costs are lower. Development of CIDP upon treatment with interferon alpha has also been reported, however.117,118


Haematopoietic Autologous Stem Cell Transplantation Autologous stem cell transplantation was used successfully in CIDP for the first time in 2001.119


Since this first case report, successful


treatment with autologous stem cell transplantation has been reported in several patients with CIDP refractory to other treatments.83,120–122


three patients with CIDP improved.123


In an open label non-randomised study, two of One patient relapsed five years


after transplantation. This patient was no longer refractory to treatment and responded well to normal doses of IVIg.124


One case report describes a patient who developed CIDP after autologous stem EUROPEAN NEUROLOGICAL REVIEW


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